Researchers have revealed the previously unknown function of the FAM126A gene in supporting myelination (the formation of the myelin sheath that protects neurons), a critically important process in the development and recovery of acute exacerbations in multiple sclerosis (MS). The research paper, entitled “The leukodystrophy protein FAM126A (hyccin) regulates PtdIns(4)P synthesis at the plasma membrane,” was published in the journal Nature Cell Biology.
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